Kim et al. 2019 (PRJNA532897)
General Details
Title | Next Generation Sequencing Facilitates Quantitative Analysis of Wild Type and FMCD mice Translatomics |
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Organism | |
Number of Samples | 9 |
Release Date | 2019/04/15 00:00 |
Sequencing Types | |
Protocol Details |
Study Links
GWIPS-viz | Trips-Viz |
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Repository Details
SRA | SRP192590 |
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ENA | SRP192590 |
GEO | GSE129818 |
BioProject | PRJNA532897 |
Publication
Title | |
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Authors | Kim JK,Cho J,Kim SH,Kang HC,Kim DS,Kim VN,Lee JH |
Journal | The Journal of clinical investigation |
Publication Date | 2019 Oct 1 |
Abstract | Brain somatic mutations confer genomic diversity in the human brain and cause neurodevelopmental disorders. Recently, brain somatic activating mutations in MTOR have been identified as a major etiology of intractable epilepsy in patients with cortical malformations. However, the molecular genetic mechanism of how brain somatic mutations in MTOR cause intractable epilepsy has remained elusive. In this study, translational profiling of intractable epilepsy mouse models with brain somatic mutations and genome-edited cells revealed a novel translational dysregulation mechanism and mTOR activation-sensitive targets mediated by human MTOR mutations that lead to intractable epilepsy with cortical malformation. These mTOR targets were found to be regulated by novel mTOR-responsive 5'-UTR motifs, distinct from known mTOR inhibition-sensitive targets regulated by 5' terminal oligopyrimidine motifs. Novel mTOR target genes were validated in patient brain tissues, and the mTOR downstream effector eIF4E was identified as a new therapeutic target in intractable epilepsy via pharmacological or genetic inhibition. We show that metformin, an FDA-approved eIF4E inhibitor, suppresses intractable epilepsy. Altogether, the present study describes translational dysregulation resulting from brain somatic mutations in MTOR, as well as the pathogenesis and potential therapeutic targets of intractable epilepsy. |
PMC | PMC6763223 |
PMID | 31483294 |
DOI |
Run Accession | Study Accession | Scientific Name | Cell Line | Library Type | Treatment | GWIPS-viz | Trips-Viz | Reads | BAM | BigWig (F) | BigWig (R) | ||
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SRR8899091 | PRJNA532897 | Mus musculus | Cortical neuron sorted from brain | Ribo-Seq | |||||||||
SRR8899092 | PRJNA532897 | Mus musculus | Cortical neuron sorted from brain | Ribo-Seq | |||||||||
SRR8899093 | PRJNA532897 | Mus musculus | Cortical neuron sorted from brain | Ribo-Seq | |||||||||
SRR8899094 | PRJNA532897 | Mus musculus | Cortical neuron sorted from brain | Ribo-Seq | |||||||||
SRR8899095 | PRJNA532897 | Mus musculus | Cortical neuron sorted from brain | Ribo-Seq | |||||||||
SRR8899096 | PRJNA532897 | Mus musculus | Cortical neuron sorted from brain | Ribo-Seq | |||||||||
SRR8899097 | PRJNA532897 | Mus musculus | Cortical neuron sorted from brain | Ribo-Seq | |||||||||
SRR8899089 | PRJNA532897 | Mus musculus | Cortical neuron sorted from brain | Ribo-Seq | |||||||||
SRR8899090 | PRJNA532897 | Mus musculus | Cortical neuron sorted from brain | Ribo-Seq | |||||||||
Run Accession | Study Accession | Scientific Name | Cell Line | Library Type | Treatment | GWIPS-viz | Trips-Viz | Reads | BAM | BigWig (F) | BigWig (R) |
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